About this article: Source GLP-1 does not accept payment to feature or omit research. This summary is compiled directly from published pharmacovigilance and registry literature; see full citations at the end.

Randomized controlled trials establish whether a drug works and is reasonably safe under controlled, monitored conditions with a defined population. They are not well-suited to catching rare adverse events, understanding how a drug performs across the full diversity of real-world patients, or tracking what actually happens once a medication scales to millions of users outside a trial's structure. That's the specific gap real-world evidence registries and pharmacovigilance databases are built to fill — and for GLP-1 medications, several distinct data sources are now generating a genuinely large post-marketing evidence base.

What the major data sources actually are

Safety signals that emerged post-marketing

A February 2026 narrative review synthesizing FDA and EMA regulatory databases alongside pharmacovigilance reports identified rare but serious post-marketing safety signals including acute pancreatitis, intestinal obstruction, thyroid C-cell hyperplasia, acute kidney injury, and transient worsening of diabetic retinopathy.[1] More recent reports from 2023–2025 identified additional signals — perioperative aspiration risk, neuropsychiatric events, and mild tachycardia — that prompted updated product labeling.[1]

36% / 47% one-year adherence in a 16M+ patient commercial claims study, Wegovy vs. Ozempic initiators
14.3% still on therapy at two years, per a follow-on persistence study
Varies by molecule — FAERS analysis found lixisenatide had the strongest hypoglycemia association of six GLP-1RAs studied

A specific example: what a real registry study looks like, and its limits

A real-world pharmacovigilance analysis comparing gynecological hemorrhagic events between tirzepatide and semaglutide, using FAERS data from 2022–2025, found no disproportionate signal indicating tirzepatide carries greater risk than semaglutide for these events.[2] But the study's own methodology section flagged a specific limitation worth understanding broadly: 94.6% of tirzepatide adverse event reports came directly from consumers, versus 53.4% for semaglutide — a substantial reporting-source disparity that complicates direct comparison, since consumer-submitted reports and clinician-submitted reports often differ in detail and follow-up completeness.

Why this methodological detail matters generally

FAERS-based disproportionality analyses are useful hypothesis-generating tools, not incidence measurements — there's no reliable denominator of how many total patients were exposed to each drug, and reporting behavior itself can vary by drug, time period, media attention, and reporter type. A "no signal detected" finding in FAERS means the data didn't show a disproportionate reporting pattern; it does not mean a risk has been ruled out with the same confidence a randomized trial would provide.

An unexpected finding from a broader phenome-wide study

A study using the All of Us Research Program (n=18,746 adults with type 2 diabetes) compared a wide range of downstream diagnoses following GLP-1RA prescription against SGLT2 inhibitor and DPP-4 inhibitor prescriptions.[3] While prior research had reported reduced substance use and psychotic disorder diagnoses among GLP-1RA users, this analysis found an increased risk of dysthymic disorder diagnosis relative to SGLT2 inhibitor users (intention-to-treat hazard ratio 2.30, 95% CI 1.53–3.46) — a specific, quantified finding that illustrates why phenome-wide registry approaches can surface signals neither trials nor narrower safety studies were designed to detect.[3]

The long-term effectiveness of GLP-1s outside controlled trials remains unclear.

Why adherence data matters as much as safety data

Registry-based persistence data has consistently found real-world adherence substantially below what trial completion rates would suggest — a 2021 commercial claims study of over 16 million people found only 36% one-year adherence for Wegovy initiators and 47% for Ozempic; a 2025 follow-on study found just 14.3% remained on therapy at two years.[4] This matters clinically because most of the dramatic weight-loss figures reported in trials reflect sustained use over the full trial period — real-world effectiveness at a population level depends heavily on how many patients actually remain on treatment long enough to reach those outcomes.

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Registry studies generally cannot establish causation with the same confidence as randomized trials. Patients who discontinue a medication, or who are prescribed one drug over another, differ systematically from those who don't — confounding that propensity matching and other statistical techniques can reduce but not eliminate entirely.

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